The face of the healthy people around them is a sign of terror, when the novel attacks the corona virus. Especially in Europe, developed countries; The virus has become a global epidemic in the world with its health system. Those who survive, as long as they are alive, witness a global epidemic, which is called 'pandemic' in English. Scientists, researchers, even ordinary people ask the question : how is this virus so successful? Not a deadly disease, but it is not clear at first whether or not he has been infected; How is the virus taking the lives of thousands of people?
The World Health Organisation and various research institutes are regularly reporting the epidemic groundswells around the world; Image source: WHO
Coronavirus, a family of viruses. Each member's virus is in the particulate shards with a little genetic material (RNA or ribo nucleic acid) and a lipid envelope that is used to cover the genetic material. Scientists have named the virus sars-CoV-2, the sars-cov of the same family, and MERS-CoV, the virus that caused the global covid-19 epidemic, but none have been successful in global attacks.
Although the lipid cover of the virus is highly sensitive to soap, the genetic material is not likely to be transmitted when the lipid cover is damaged by the soap. The virus can be active outside for three or four hours in the outer shell of wood, paper, cloth, steel or plastic. However, some studies suggest that the virus can be active for one to three days, meaning that if you and I touch that place, the virus may be in our hands. However, the virus is not able to grow outside the body, meaning that if it cannot reach the body in this time, its play is over!
How does this virus enter the body?
This Covid-19 is not a common flu, its symptoms and types are different from those of the rest of the corona viruses. Four members of the Corona virus family (OC43, HKU1, NL63, 229E) were responsible for one-third of the common cold fever worldwide, the other two were more severe than the Covid-19, according to the signs of SARS and mars.
But none of this could shape the global epidemic. The 7th member of this family (SARS-CoV-2) is now moving the world.
The formation of this virus indicates its success. Like a ball with numerous thorns, these thorns are called spikes. These spikes can be linked to ace2, a protein in our cells. This protein is found in our cells. The virus's spike in our protein is the first step in the virus's human infection. Research says that the virus (now also known as the SARS Classic) may be more tightly linked to ACE2 than the previous ONES virus. "This strong bond is very important for spreading from one person to another, and very few viruses are enough to start the infection because of strong bonds," says Columbia University researcher Angela Rasmussen. "
The spikes in the virus's outer shell are composed of two pairs of parts. It is activated only when the two parts are separated, and the virus can then enter our cells. The separation was very difficult in the SARS classic (which was responsible for the sars) virus. But this time, an enzyme called Furin works to isolate the two parts of the virus responsible for covid-19, and unfortunately, human cells can produce this furin enzyme itself and furin enzymes are found in our cells. The most important thing is the unusual phenomenon that has been observed in the virus, says Christian Anderson, a researcher at the Scripps Research Transnational Institute.
The virus that attacks human snout is called the respirator virus. The SARS-CoV-2 virus of Covid-19 is also a respirator ya virus. However, there is a specific place to attack these viruses. For example, some respiratory viruses attack the upper part of our immune system. They spread very quickly through sneezing, coughing, but they do not cause much suffering to people. It spreads quickly, and it heals quickly. People don't even understand that he has been infected with the virus. And those who attack the bottom of the machine are very slow to spread from person to person, but they are very much enjoying the human being.
SARS-CoV-2 can attack the human's equipment both on and below. Scientists think that this task has been made easier because of the availability of furin. And that's why the virus has started spreading as long as it is spreading to the human body. We may be delivering the virus from one person to another.
From the information from scientists, the origin of the virus may be spread from the bat, the bat, to the pangolin (as we call the banrui), from there to humans. Virus is very efficient in this case. He can change his pet to spread from one animal to another with incredible speed.
However, the conspiracy theories are not less than the unusual features of the virus. How did this virus get properly connected to our ACE2 protein? The virus has also been found to be such a rare feature from hundreds of thousands of viruses on earth! However, scientists say genomic research still shows that the virus is less likely to be produced in the lab. Instead, the badass, the pangolin, entered the human being.
Virus mutations can cause minor changes in the genetic material. Surprisingly, however, there has been no major change in the virus since the beginning. It is also happening just as the common virus mutates. Hundreds of mutations have been recorded so far. But none is of such a large size. University of North Carolina researcher Lisa Gralinski says that if the virus is under pressure to make a big change, it is becoming more and more susceptible to infection.
But scientists are not sure about this either. A virus that was isolated from a Singaporean patient showed that the virus had lost part of its gene. The change is likely to make the virus less aggressive. However, it was not found in samples from many countries. So scientists say an exceptional phenomenon.
The way the body attacks.
The virus attacks cells carrying ACE2 in our respiratory tract after entering the body. Our respiratory cells are destroyed, while the rest of the dead cells in our respiratory tract are in trouble. If the immune system is weak, the virus reaches the lungs to destroy cells. The virus attacks celia celia in the lungs. Silya's work is like a bush. Dead cells accumulate in the lungs as the infection levels increase. As a result, the respiratory problem that was earlier manifested in the outside may require oxygen for some.
Our body's defence scare attacks the virus and causes fever. If our body's defence system can fight the virus, people can recover from the covid-19 infection. That's young, unspoilt and has a good defence system for those people. However, if someone already has lung, respiratory infections or a weak immune system (immune-compromised), it is easier to damage our lungs than to the virus. So globally, older people are a little more at risk.
However, many of the young or middle-aged people who have a good body defence are also at risk because our body defence shards are often broken. Especially when the infection of the body increases and the defence system starts to function at a higher level, the damage to the body is greater. Such a highly functional defence system creates a situation called cytokine storm in the body.
In this situation, the normal cells of the body are destroyed. The virus attack causes a lot of complications in the body due to the weak system. In a way, the broken immune system can destroy the cells of the body and damage the organs. And if the person has an infection already, the condition becomes more severe. Age here determines the ability of the defence system to determine whether the person will reach the cytokine storm.
The number of people working with the corona virus in the world for a long time is also negligible. University of Pennsylvania researcher Susan Weis has been researching the corona virus for nearly forty years. According to him, many of the researchers have started researching the corona virus since the SARS epidemic in 2002. That's a little too. So people need to start a lot of work with the corona virus. Or it is not unusual for global communication to spread like this again and again.
Ohh man that was wonderful💚