Although 11 scientists have discovered about the entire COVID-19, the epidemic also highlights the reality that we have not yet realized our immune structure. For example, one problem that does get researchers into trouble is that there are indeed all kinds of people who have caught the SARS-CoV-2 virus...not showing any symptoms or inflammatory symptoms at all. No one feels very positive about how accustomed to these asymptomatic or asymptomatic instances. This is for a disease that accounts for 5% to 30% of humans in the hospital! Although the researchers are very sure that there is no solution to why humans do not feel the results of SARS-CoV-2. In-depth study of various abilities and motivations will roughly train our body how to resist pathogens. Now, technically speaking, all people with COVID-19 start with asymptomatic development or PR symptoms. No one will immediately cough the main virus into their cells, and this virus will definitely be considered a virus and persuade it to start producing more viruses.
The incubation period is very long, ranging from a few days to two weeks before humans begin to feel the pollution. Similarly, the "feel" seems to be different. It may be milder symptoms and similar blunt taste or smell, life-threatening breathing problems, or all symptoms of coronary heart disease. Don't remember someone falling on that spectrum; even moderate signs will make the case remember asymptomatic pollution. It is also important to note that the appearance of COVID-19 without symptoms does not mean that there will be no results in a long time frame. Nevertheless, scientists are still paying attention to whether the asymptomatic infection will cause symptoms and signs. However, asymptomatic sincerity reduces the percentage of immediate emergency hospital treatment. One aspect worth mentioning is that some human companies are much less able to withstand fierce events such as young people. As an institution, people under the age of 18, especially those between the ages of two and thirteen, seem unlikely to resolve COVID-19, and if they do, they are very likely to be addicted to signs. This may be because once the child's immune device is established, novelty is its strength. Children are beginners from all over the world, so they don't have countless sick businessmen like adults.
Given that SARS-CoV-2 is new to everyone, young people seem to have some blessings that can help them clean it earlier and more correctly than they feel the signs. The integral part is that we are no longer really sure of the blessing of who we are now. There has not been enough research to study how the immune structure changes with age to accurately determine the possible role. However, due to the stimulation of COVID-19, a large number of researchers will redouble their efforts in this area, so in the next few years, we will realize a lot of additional content! That is, one factor that seems to increase with age is the distribution of ACE2 receptors, which may be particularly applicable to this virus. These are SARS-CoV-2 viruses that use these proteins to inhibit and infect our cells. They may be in the organization during the framework. But their presence inside the nasopharynx is particularly important because in this case, respiratory viruses like SARS-CoV-2 usually enter our body, and scientists also agree that these viruses can persist and reproduce.
If the content of ACE2 in a person's organization of a young person is much less, then they cannot easily obtain full-scale pollution, which is related to the feeling that they no longer have signed. Look, in May 2020, the Journal of the American Medical Association found that the pastime of the ACE2 gene gradually decreases in children and accelerates with age. Now, another study that has not yet been peer-reviewed suggests that ACE2 in the nasal cavity will drop again after the age of 60. Therefore, if this result is found, it will not explain why the organization tends to show additional extreme signs. But even in the elderly, the expression of ACE2 greatly depends on environmental factors. For example, adults with allergic respiratory reactions seem to have much less ACE2 in their nasal passages. Therefore, similar things affect which ACE2 and which ACE2 someone uses, and may also affect their likelihood of obtaining asymptomatic contamination. It is no longer ACE2. Research advocates that the virus hopes to contaminate cells with the help of all other active proteins called TMPRSS2. And young people.