Today we are going to talk about something that cannot be finished. If you write about him, it will be an essay ... !!! So today we will discuss a little about that famous person. Oh, I didn't introduce you to him, He is Mr. Thyroid. We will discuss today .... ??? Give me a moment, before we go into the discussion. Let's talk a little bit about thyroid, or he'll get a little angry. #Name: Mr. Thyroid #Location: Front of the neck, below the Adam's apple. #Artery supply: Superior, Inferior thyroid arteries. It is an endocrine gland in the neck consisting of two connected lobes. The lower two thirds of the lobes are connected by a thin band of tissue called the thyroid isthmus. Mr. Thyroid secretes 3 types of hormones: Triiodothyronine (T3) and thyroxine (T4) - and a peptide hormone, calcitonin. Let us now focus on the basics: The disease I am going to talk about is, #Sub_acute_thyroiditis. Well, we have seen synonyms, just as there are two synonyms. 1. deQuervain's thyroiditis 2. Granulomatous giant cell thyroiditis We are good at memorizing definitions from a young age (1 st year). I have even seen that long definition of public health and prof viva. So this disease should also have a definition. #Sub_acute_thyroiditis Subacute thyroiditis involves inflammation of the thyroid gland that usually follows an upper respiratory infection and then subsides. It is an uncommon condition thought to be caused by viral infection of the thyroid gland. The condition often occurs after a viral infection of the upper respiratory tract. Mumps virus, influenza virus, and other respiratory viruses have been found to cause subacute thyroiditis. It is a disease in which two characters of the thyroid are observed. Character 2 is Hyperthyroidism & Hypothyroidism. Now the question is why two opposite religious conditions arise here. We will first know why hyperthyroidism is happening: Sub acute thyroiditis is a viral infection of the thyroid gland. This infection is usually caused by Mumps virus, Coxasackie virus and Adeno virus. This infection causes transient inflammation of the thyroid follicles. The thyroid follicle is surrounded by follicular cells and has a soft colloid in the middle. This colloid contains thyroid hormone. When the thyroid follicle becomes inflamed and ruptures, the thyroid hormone that has accumulated for 3 months comes out at once. This is hyperthyroidism due to the large amount of thyroid hormone coming into the blood at the same time. This hyperthyroidism lasts up to 4-6 weeks. Well now we understand why hyperthyroidism is happening, now let's look at why hypothyroidism is happening .... As we have said before, follicular cells are inflamed. This follicular cell is being damaged and ruptured as a result of inflammation. Another important thing is that this thyroid hormone is synthesized from follicular cells. Therefore, due to its inflammation, it can no longer synthesize hormones. As a result, when the extra hormones are used for rupture, no new thyroid hormone comes into the blood. This leads to the onset of hypothyroidism. At this moment, we understand why these two different conditions came about. But we need to know how long this condition will remain in our body. We have said before that this stage lasts for 4-6 months. Then these follicular cells begin to regenerate themselves again. Then over time, the hormone synthesis begins again. Now we will focus on the most important point before going to treatment is how the patient will present his condition. Symptoms: 1. Pain in the front of the neck. 2. Tenderness when gentle pressure is applied to the thyroid gland (palpation). 3. Fever 4. Weakness 5. Fatigue There are also some more conditions. Now we will give some lab test to the patient. Laboratory tests in the early phase of the disease may reveal: High serum thyroglobulin level Low radioactive iodine uptake Low serum thyroid stimulating hormone (TSH) level High serum free T4 (thyroid hormone, thyroxine) level High e
